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Science 13 July 2007:
Vol. 317. no. 5835, pp. 190 - 191
DOI: 10.1126/science.317.5835.190
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News Focus

NEUROSCIENCE:
Autism's Cause May Reside in Abnormalities at the Synapse

Ken Garber

New genetic evidence is leading researchers to home in on the cleft separating neurons as the site where the disorder may originate.

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THIS ARTICLE HAS BEEN CITED BY OTHER ARTICLES:
Mutations in the calcium-related gene IL1RAPL1 are associated with autism.
A. Piton, J. L. Michaud, H. Peng, S. Aradhya, J. Gauthier, L. Mottron, N. Champagne, R. G. Lafreniere, F. F. Hamdan, S2D team, et al. (2008)
Hum. Mol. Genet. 17, 3965-3974
   Abstract »    Full Text »    PDF »
Synaptic Imbalance, Stereotypies, and Impaired Social Interactions in Mice with Altered Neuroligin 2 Expression.
R. M. Hines, L. Wu, D. J. Hines, H. Steenland, S. Mansour, R. Dahlhaus, R. R. Singaraja, X. Cao, E. Sammler, S. G. Hormuzdi, et al. (2008)
J. Neurosci. 28, 6055-6067
   Abstract »    Full Text »    PDF »
A Neuroligin-3 Mutation Implicated in Autism Increases Inhibitory Synaptic Transmission in Mice.
K. Tabuchi, J. Blundell, M. R. Etherton, R. E. Hammer, X. Liu, C. M. Powell, and T. C. Sudhof (2007)
Science 318, 71-76
   Abstract »    Full Text »    PDF »


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Science. ISSN 0036-8075 (print), 1095-9203 (online)