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A cytosolic protein that translocates into the mitochondria may serve as an integration point for signaling pathways that control longevity and cell death.
The authors are in the Department of Pathology, Anatomy and Cell Biology, Thomas Jefferson University, Philadelphia, PA 19107, USA. E-mail: gyorgy.hajnoczky{at}jefferson.edu, jan.hoek{at}jefferson.edu
Mitochondrial Complex I Defect Induces ROS Release and Degeneration in Trabecular Meshwork Cells of POAG Patients: Protection by Antioxidants.
Y. He, K. W. Leung, Y.-H. Zhang, S. Duan, X.-F. Zhong, R.-Z. Jiang, Z. Peng, J. Tombran-Tink, and J. Ge (2008)
Invest. Ophthalmol. Vis. Sci.
49, 1447-1458
|Abstract »|Full Text »|PDF »
Synergy between docosahexaenoic acid and butyrate elicits p53-independent apoptosis via mitochondrial Ca2+ accumulation in colonocytes.
S. S. N. Kolar, R. Barhoumi, E. S. Callaway, Y.-Y. Fan, N. Wang, J. R. Lupton, and R. S. Chapkin (2007)
Am J Physiol Gastrointest Liver Physiol
293, G935-G943
|Abstract »|Full Text »|PDF »
