Note to users. If you're seeing this message, it means that your browser cannot find this page's style/presentation instructions -- or possibly that you are using a browser that does not support current Web standards. Find out more about why this message is appearing, and what you can do to make your experience of our site the best it can be.
How a key immunological regulator prevents autoimmune disease has been unclear. Live imaging shows that it may prevent immune cells from lingering too long in the lymph nodes.
The author is in the Program on Inflammatory Disease Research, Infectious and Inflammatory Disease Center, and in the Program of Signal Transduction, Cancer Center, The Burnham Institute for Medical Research, La Jolla, CA 92037, USA. E-mail: tmustelin{at}burnham.org
The editors suggest the following Related Resources on Science sites:
In Science Magazine
REPORTS
Helga Schneider, Jos Downey, Andrew Smith, Bernd H. Zinselmeyer, Catherine Rush, James M. Brewer, Bin Wei, Nancy Hogg, Paul Garside, and Christopher E. Rudd (29 September 2006) Science313 (5795), 1972.
[DOI: 10.1126/science.1131078] |Abstract »|Full Text »|PDF »|Supporting Online Material »
THIS ARTICLE HAS BEEN CITED BY OTHER ARTICLES:
A CD26-Controlled Cell Surface Cascade for Regulation of T Cell Motility and Chemokine Signals.
Z. Liu, M. Christensson, A. Forslow, I. De Meester, and K.-G. Sundqvist (2009)
J. Immunol.
183, 3616-3624
|Abstract »|Full Text »|PDF »
{alpha}1-Antitrypsin monotherapy induces immune tolerance during islet allograft transplantation in mice.
E. C. Lewis, M. Mizrahi, M. Toledano, N. DeFelice, J. L. Wright, A. Churg, L. Shapiro, and C. A. Dinarello (2008)
PNAS
105, 16236-16241
|Abstract »|Full Text »|PDF »
