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Science 7 June 1996:
Vol. 272. no. 5267, pp. 1422 - 0
DOI: 10.1126/science.272.5267.1422
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Christine Mlot

The bacteria Chlamydia has always been an odd bug, causing an illness in parrots as well as sexually transmitted diseases. Now a recently discovered species, C. pneumoniae, offers an even bigger surprise: It consistently turns up in the arteries of patients with atherosclerosis.


THIS ARTICLE HAS BEEN CITED BY OTHER ARTICLES:
Chlamydia pneumoniae Induces Tissue Factor Expression in Mouse Macrophages via Activation of Egr-1 and the MEK-ERK1/2 Pathway.
F. Bea, M. H. Puolakkainen, T. McMillen, F. N. Hudson, N. Mackman, C. C. Kuo, L. A. Campbell, and M. E. Rosenfeld (2003)
Circ. Res. 92, 394-401
   Abstract »    Full Text »    PDF »
Comparison of whole genome sequences of Chlamydia pneumoniae J138 from Japan and CWL029 from USA.
M. Shirai, H. Hirakawa, M. Kimoto, M. Tabuchi, F. Kishi, K. Ouchi, T. Shiba, K. Ishii, M. Hattori, S. Kuhara, et al. (2000)
Nucleic Acids Res. 28, 2311-2314
   Abstract »    Full Text »    PDF »
Chlamydia trachomatis (Mouse Pneumonitis Strain) Induces Cardiovascular Pathology following Respiratory Tract Infection.
Y. Fan, S. Wang, and X. Yang (1999)
Infect. Immun. 67, 6145-6151
   Abstract »    Full Text »    PDF »
Chlamydia pneumoniae Infection of Vascular Smooth Muscle and Endothelial Cells Activates NF-{kappa}B and Induces Tissue Factor and PAI-1 Expression : A Potential Link to Accelerated Arteriosclerosis.
R. Dechend, M. Maass, J. Gieffers, R. Dietz, C. Scheidereit, A. Leutz, and D. C. Gulba (1999)
Circulation 100, 1369-1373
   Abstract »    Full Text »    PDF »


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