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Science 11 August 2006:
Vol. 313. no. 5788, pp. 767 - 768
DOI: 10.1126/science.1131714
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Perspectives

IMMUNOLOGY:
When F-actin Becomes Too Much of a Good Thing

Michael L. Dustin

An excess of a protein that regulates actin assembly in white blood cells unexpectedly kills the cell by altering mitochondrial function.

The author is in the Progam in Molecular Pathogenesis, Skirball Institute, New York University Medical Center, 540 First Avenue, New York, NY 10016, USA. E-mail: dustin{at}saturn.med.nyu.edu

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Science. ISSN 0036-8075 (print), 1095-9203 (online)