Note to users. If you're seeing this message, it means that your browser cannot find this page's style/presentation instructions -- or possibly that you are using a browser that does not support current Web standards. Find out more about why this message is appearing, and what you can do to make your experience of our site the best it can be.

Science 24 October 1969:
Vol. 166. no. 3904, pp. 524 - 525
DOI: 10.1126/science.166.3904.524
Prev | Table of Contents | Next

Articles

Axonal Transport of Proteins in Experimental Neuropathies

David E. Pleasure 1, Kathleen C. Mishler 1, and W. King Engel 1

1 Medical Neurology Branch, National Institute of Neurological Diseases and Stroke, Bethesda, Maryland 20014

Axoplasmic flow of proteins is interrlupted in cats with neuropatlhy induced by acrylamide, but it is niot initerrupted in nzormlal cats and in those with neuropathy induced by tri-orthocresyl phiospliate. The proteins move from lumbosacral motor neurons along the ventral roots and from ganglion cells toward the spinal cord along the dorsal roots at about 1frac12 millimeters per day.


THIS ARTICLE HAS BEEN CITED BY OTHER ARTICLES:
Axonal Transport of Dopamine-beta-Hydroxylase by Human Sural Nerves in vitro.
S. Brimijoin, P. Capek, and P. J. Dyck (1973)
Science 180, 1295-1297
   Abstract »    PDF »
Peripheral Nerve Changes in Thiamine-Deficient Rats: An Electron Microscope Study.
J. Prineas (1970)
Arch Neurol 23, 541-548
   Abstract »    PDF »


To Advertise     Find Products

Science. ISSN 0036-8075 (print), 1095-9203 (online)