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Science 15 July 1994: Vol. 265. no. 5170, p. 320 DOI: 10.1126/science.8023154
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Articles
CMV-p53 interaction may help explain clogged arteries
J Marx
No abstract or summary view of this item is available. To view the full-text version, follow this link.
Article topics:
- Medicine..Genes
- Physiology..Coronary Arteries
- Physiology..Smooth Muscle
- Oncology..p53
- Medicine..Angioplasty
THIS ARTICLE HAS BEEN CITED BY OTHER ARTICLES:
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- P. M. Krosky, K. Z. Borysko, M. R. Nassiri, R. V. Devivar, R. G. Ptak, M. G. Davis, K. K. Biron, L. B. Townsend, and J. C. Drach (2002)
Antimicrob. Agents Chemother.
46, 478-486
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- p53: The Challenge of Linking Basic Science and Patient Management.
- P. A. Hall (1998)
Oncologist
3, 218-224
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- The Challenge of p53: Linking Biochemistry, Biology, and Patient Management.
- S. E. Bray, C. Schorl, and P. A. Hall (1998)
Stem Cells
16, 248-260
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- Atherosclerosis in Marek's disease virus infected hypercholesterolemic roosters is reduced by HMGCoA reductase and ACE inhibitor therapy.
- A. Lucas, E. Dai, L.-Y. Liu, and P. N Nation (1998)
Cardiovasc Res
38, 237-246
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- Remodeling Rather Than Neointimal Formation Explains Luminal Narrowing After Deep Vessel Wall Injury : Insights From a Porcine Coronary (Re)stenosis Model.
- H. Rud Andersen, M. Mæng, M. Thorwest, and E. Falk (1996)
Circulation
93, 1716-1724
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- Gene therapy for clogged arteries passes test in pigs.
- M Barinaga (1994)
Science
265, 738
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