Science 15 April 1994: Vol. 264. no. 5157, pp. 365 - 367 DOI: 10.1126/science.8153620

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Articles

Early Treatment with Hydroxyethyl Starch 130/0.4 Causes Greater Inhibition of Pulmonary Capillary Leakage and Inflammatory Response than Treatment Instituted Later in Sepsis Induced by Cecal Ligation and Puncture in Rats.

X. Feng, Y. Hu, J. Ding, Y. Ge, J. Song, Q. Ai, Z. Zhang, and J. Xu (2007)
Ann. Clin. Lab. Sci. 37, 49-56
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A Soluble Form of the Triggering Receptor Expressed on Myeloid Cells-1 Modulates the Inflammatory Response in Murine Sepsis.

S. Gibot, M.-N. Kolopp-Sarda, M.-C. Bene, P.-E. Bollaert, A. Lozniewski, F. Mory, B. Levy, and G. C. Faure (2004)
J. Exp. Med. 200, 1419-1426
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CD137-Deficient Mice Have Reduced NK/NKT Cell Numbers and Function, Are Resistant to Lipopolysaccharide-Induced Shock Syndromes, and Have Lower IL-4 Responses.

D. S. Vinay, B. K. Choi, J. S. Bae, W. Y. Kim, B. M. Gebhardt, and B. S. Kwon (2004)
J. Immunol. 173, 4218-4229
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Characterization of the Systemic Loss of Dendritic Cells in Murine Lymph Nodes During Polymicrobial Sepsis.

P. A. Efron, A. Martins, D. Minnich, K. Tinsley, R. Ungaro, F. R. Bahjat, R. Hotchkiss, M. Clare-Salzler, and L. L. Moldawer (2004)
J. Immunol. 173, 3035-3043
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Overexpression of Interleukin-15 Increases Susceptibility to Lipopolysaccharide-Induced Liver Injury in Mice Primed with Mycobacterium bovis Bacillus Calmette-Guerin.

T. Yajima, H. Nishimura, K. Saito, H. Kuwano, and Y. Yoshikai (2004)
Infect. Immun. 72, 3855-3862
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Regulation of Kinase Cascades and Transcription Factors by a Poly(ADP-Ribose) Polymerase-1 Inhibitor, 4-Hydroxyquinazoline, in Lipopolysaccharide-Induced Inflammation in Mice.

B. Veres, B. Radnai, F. Gallyas Jr., G. Varbiro, Z. Berente, E. Osz, and B. Sumegi (2004)
J. Pharmacol. Exp. Ther. 310, 247-255
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{beta}2-Adrenoceptor Activation Attenuates Endotoxin-Induced Acute Renal Failure.

A. Nakamura, A. Imaizumi, Y. Yanagawa, T. Kohsaka, and E. J. Johns (2004)
J. Am. Soc. Nephrol. 15, 316-325
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The Pathophysiology and Treatment of Sepsis.

R. S. Hotchkiss and I. E. Karl (2003)
N. Engl. J. Med. 348, 138-150
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Jaks, STATs, Cytokines, and Sepsis.

M. J. Scott, C. J. Godshall, and W. G. Cheadle (2002)
Clin. Vaccine Immunol. 9, 1153-1159
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Neutrophil-Kupffer cell interaction: a critical component of host defenses to systemic bacterial infections.

S. H. Gregory and E. J. Wing (2002)
J. Leukoc. Biol. 72, 239-248
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Apoptosis in sepsis: a new target for therapeutic exploration.

C. OBERHOLZER, A. OBERHOLZER, M. CLARE-SALZLER, and L. L. MOLDAWER (2001)
FASEB J 15, 879-892
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Synthesis and Surface Expression of CD14 by Human Endothelial Cells.

H. P. A. Jersmann, C. S. T. Hii, G. L. Hodge, and A. Ferrante (2001)
Infect. Immun. 69, 479-485
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Endotoxin-Induced Mortality Is Related to Increased Oxidative Stress and End-Organ Dysfunction, Not Refractory Hypotension, in Heme Oxygenase-1-Deficient Mice.

P. Wiesel, A. P. Patel, N. DiFonzo, P. B. Marria, C. U. Sim, A. Pellacani, K. Maemura, B. W. LeBlanc, K. Marino, C. M. Doerschuk, et al. (2000)
Circulation 102, 3015-3022
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Molecular mechanisms of NF-{kappa}B activation induced by bacterial lipopolysaccharide through Toll-like receptors.

Guolong Zhang and S. Ghosh (2000)
Innate Immunity 6, 453-457
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Involvement of the Acute Phase Protein alpha 1-Acid Glycoprotein in Nonspecific Resistance to a Lethal Gram-negative Infection.

T. Hochepied, W. Van Molle, F. G. Berger, H. Baumann, and C. Libert (2000)
J. Biol. Chem. 275, 14903-14909
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p53-Dependent and -Independent Pathways of Apoptotic Cell Death in Sepsis.

R. S. Hotchkiss, K. W. Tinsley, J.-J. Hui, K. C. Chang, P. E. Swanson, A. M. Drewry, T. G. Buchman, and I. E. Karl (2000)
J. Immunol. 164, 3675-3680
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Prevention of lymphocyte cell death in sepsis improves survival in mice.

R. S. Hotchkiss, K. W. Tinsley, P. E. Swanson, K. C. Chang, J. P. Cobb, T. G. Buchman, S. J. Korsmeyer, and I. E. Karl (1999)
PNAS 96, 14541-14546
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Inhibition of NF-{kappa}B Activation by Pyrrolidine Dithiocarbamate Prevents In Vivo Expression of Proinflammatory Genes.

S. F. Liu, X. Ye, and A. B. Malik (1999)
Circulation 100, 1330-1337
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Moesin Functions as a Lipopolysaccharide Receptor on Human Monocytes.

Z. N. Tohme, S. Amar, and T. E. Van Dyke (1999)
Infect. Immun. 67, 3215-3220
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Overexpression of Bcl-2 in Transgenic Mice Decreases Apoptosis and Improves Survival in Sepsis.

R. S. Hotchkiss, P. E. Swanson, C. M. Knudson, K. C. Chang, J. P. Cobb, D. F. Osborne, K. M. Zollner, T. G. Buchman, S. J. Korsmeyer, and I. E. Karl (1999)
J. Immunol. 162, 4148-4156
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Lipoprotein Release by Bacteria: Potential Factor in Bacterial Pathogenesis.

H. Zhang, D. W. Niesel, J. W. Peterson, and G. R. Klimpel (1998)
Infect. Immun. 66, 5196-5201
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Tumor Necrosis Factor Alpha Transcription in Macrophages Is Attenuated by an Autocrine Factor That Preferentially Induces NF-kappa B p50.

M. Baer, A. Dillner, R. C. Schwartz, C. Sedon, S. Nedospasov, and P. F. Johnson (1998)
Mol. Cell. Biol. 18, 5678-5689
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Platelet- and macrophage-derived endogenous cannabinoids are involved in endotoxin-induced hypotension.

K. Varga, J. A. Wagner, D. T. Bridgen, and G. Kunos (1998)
FASEB J 12, 1035-1044
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Lipopolysaccharide-Related Stimuli Induce Expression of the Secretory Leukocyte Protease Inhibitor, a Macrophage-Derived Lipopolysaccharide Inhibitor.

F. Jin, C. F. Nathan, D. Radzioch, and A. Ding (1998)
Infect. Immun. 66, 2447-2452
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Therapeutic Effects of Nitric Oxide Inhibition during Experimental Fecal Peritonitis: Role of Interleukin-10 and Monocyte Chemoattractant Protein 1.

C. M. Hogaboam, M. L. Steinhauser, H. Schock, N. Lukacs, R. M. Strieter, T. Standiford, and S. L. Kunkel (1998)
Infect. Immun. 66, 650-655
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Transgenic mice expressing rabbit C-reactive protein are resistant to endotoxemia.

D. Xia and D. Samols (1997)
PNAS 94, 2575-2580
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Cytokine Therapy in Septic Shock.

J. S. Friedland, J. C. Porter, and G. P. Chrousos (1995)
N. Engl. J. Med. 333, 942-943
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Interactions of linear dicationic molecules with lipid A: structural requisites for optimal binding affinity.

S.A. David, V.I. Mathan, and P. Balaram (1995)
Innate Immunity 2, 325-336
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Hepatocytes Contribute to Soluble CD14 Production, and CD14 Expression Is Differentially Regulated in Hepatocytes and Monocytes.

Z. Pan, L. Zhou, C. J. Hetherington, and D.-E. Zhang (2000)
J. Biol. Chem. 275, 36430-36435
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The Phosphatidylinositol 3-Kinase Pathway Selectively Controls sIL-1RA Not Interleukin-1beta Production in the Septic Leukocytes.

C. A. Learn, M. S. Boger, L. Li, and C. E. McCall (2001)
J. Biol. Chem. 276, 20234-20239
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