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Science 28 August 1981:
Vol. 213. no. 4511, pp. 1030 - 1032
DOI: 10.1126/science.7268406

Articles

Science, Vol 213, Issue 4511, 1030-1032
Copyright © 1981 by American Association for the Advancement of Science


articles

Periodicity of sleep states is altered in infants at risk for the sudden infant death syndrome

RM Harper, B Leake, H Hoffman, DO Walter, T Hoppenbrouwers, J Hodgman, and MB Sterman

The normal succession of sleep and waking states through a night is disturbed in infants at risk for the sudden infant death syndrome. Compared with normal infants, siblings of the sudden infant death syndrome victims have longer intervals between active sleep epochs at particular times during the night in the newborn period and a decreased tendency to enter short waking periods at 2 and 3 months of age. The latter finding is interpreted as an increased tendency to remain asleep, or a relative failure to arouse from sleep in infants at risk.


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nCPAP improves abnormal autonomic function in at-risk-for-SIDS infants with OSA.
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J Appl Physiol 95, 1591-1597
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Altered Autonomic Function and Reduced Arousability in Apparent Life-Threatening Event Infants with Obstructive Sleep Apnea.
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Am. J. Respir. Crit. Care Med. 165, 1048-1054
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Sudden Infant Death Syndrome and Other Causes of Infant Mortality . Diagnosis, Mechanisms, and Risk for Recurrence in Siblings.
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Am. J. Respir. Crit. Care Med. 164, 346-357
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Catecholamine Neurons Alteration in the Brainstem of Sudden Infant Death Syndrome Victims.
T. Obonai, M. Yasuhara, T. Nakamura, and S. Takashima (1998)
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Infant Arousals During Mother-Infant Bed Sharing: Implications for Infant Sleep and Sudden Infant Death Syndrome Research.
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Behaviour and physiological responses during prone and supine sleep in early infancy.
B T Skadberg and T Markestad (1997)
Arch. Dis. Child. 76, 320-324
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Possible Role of the Brain Stem in Sudden Infant Death Syndrome.
N. Baba, J. J. Quattrochi, C. B. Reiner, W. Adrion, P. T. McBride, and A. J. Yates (1983)
JAMA 249, 2789-2791
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