Note to users. If you're seeing this message, it means that your browser cannot find this page's style/presentation instructions -- or possibly that you are using a browser that does not support current Web standards. Find out more about why this message is appearing, and what you can do to make your experience of our site the best it can be.

Site Tools

  • AAAS
  • Subscribe
  • Feedback

Site Search

Search Advanced

Science 19 May 1972:
Vol. 176. no. 4036, pp. 800 - 803
DOI: 10.1126/science.176.4036.800
Prev | Table of Contents | Next

Articles

Cribriform Degeneration (cri): A New Recessive Neurological Mutation in the Mouse

Margaret C. Green 1, Richard L. Sidman 2, and Omar H. Pivetta 3

1 Jackson Laboratory, Bar Harbor, Maine 04609
2 Department of Neuropathology, Harvard Medical School, Boston, Massachusetts 02115
3 Jackson Laboratory

The mutation cribriform degeneration (cri) occurred in the DBA/2J strain; it is in linkage group VIII, 31 recombination units from b. Homozygotes show severe vacuolar degeneration in white and gray matter of the spinal cord and brainstem, normocytic anemia at birth which decreases in severity with age, and abnormalities of electrolyte distribution.


THIS ARTICLE HAS BEEN CITED BY OTHER ARTICLES:
Hexachlorophene Lesions in Newborn Infants.
J. M. Gowdy, A. G. Ulsamer, J. S. Drage, T. Fujikura, W. Cockran, R. O. Fisch, W. Clark Jr, C. Zelson, M. Yates, R. V. Weile, et al. (1976)
Arch Pediatr Adolesc Med 130, 247-250
   Abstract »    PDF »


To Advertise     Find Products

Science. ISSN 0036-8075 (print), 1095-9203 (online)